That one doesn’t make sense to me either. But the notion that PUFA’s new to the human diet over the past 180 years might be able to cause trouble does make sense. I watched a lecture by Tucker Goodrich about seed oils that seemed over-the-top, but then Chris Knobbe gave a lecture at a Low Carb Down Under event on the same topic, but with actual data in support, that made a lot more sense. What Nina Teicholz has to say about PUFA’s in fast food restaurants is scary but well-documented.
Well, I think Tucker Goodrich did get harmed by PUFAs. But not everyone has the same effect.
But there are a lot of folks that believe PUFAs = obesity, Dr. Eades, Peter D. from Hyperlipid, Brad from Fire in a Bottle, etc. But I can’t tell any effect. It would be so much easier if I ate saturated fat and got an effect. But I don’t.
I do think too many PUFAs could be an issue, and of course over-heating them is troubling. Heart issues and PUFAs are particularly troubling.
It can’t possibly be that all PUFA’s are damaging, because there are some that occur naturally in the foods we evolved to eat. As I understand Dr. Knobbe and some things Nina Teicholz has reported, the problem comes from (a) PUFA’s new to the body, and (b) damage to said PUFA’s from heating oils to cook with.
It’s not for nothing that humanity’s preferred cooking fats were always butter, tallow, and lard, even in those countries that started cooking with olive oil a century or two ago. In ancient times, olive oil was strictly a cosmetic and a lamp fuel.
Olive oil can at least be cold pressed. The seed oils require as much refining as petroleum products.
Am watching an interview with Benjamin Bikman (it’s been linked in a couple of threads already, so I won’t link it again), in which he states that anything that causes systemic inflammation causes insulin resistance. One example, of course, is ω-6 fatty acids (particularly linoleic acid, the most easily oxidised fat) which are particularly abundant in the industrial seed oils. Another reason to avoid these oils.
He also states (for which I’d like to see some data) that “protein and fat combined are more anabolic than protein alone.”
I think you and I are on the same page. But let me lay out part of the anti-PUFA theory.
Allow me to put on my anti-PUFA hat. It’s true that all PUFAs aren’t bad. You refer to butter, tallow, and lard. There’s also chicken fat, a staple with some.
But what’s happened over time? I read study that was old and was looking at the fats in animals eaten on an island country. The fat in chickens was 70% saturated. 70%! The fat in their grazing/feral pigs was super high in saturated fat, too.
What would happen now, if you took your pig fat or chicken fat and had it tested? It will be quite low on saturated fat, as pigs and chickens are what they eat, and they are eating corn:
I know you like bacon. The bacon you are eating is not the bacon you would have eaten 100+ years ago. It’s filled with PUFAs, and it wasn’t then.
Add chicken to that, with these crazy chickens that are only alive for a short time, and fed higher fat (soy, corn) diets because they get bigger faster.
Then consider that basically everything – and I mean EVERYTHING – that’s prepared and that you buy at the store has some type of seed oil in it. Heck, the olives I used to buy from the local store were in sunflower oil.
We haven’t even gotten to “highly processed” foods. Almost all of those are filled with PUFAs and seed oils.
Then, you go to dinner and anything fried is fried in seed oil. Even that steak you have that’s pan fried, it’s fried in seed oil. That salad dressing is probably seed oil. Any mayo, seed oil.
You can look at something like this:
And see that we’re getting overwhelmed by seed oils.
And then you see studies where the half-life of PUFAs in your fat cells is 2+ years. Years. That means if you eat a zero PUFA diet (not possible, as I’m sure you know), it takes 2 years to go from 100 to 50 (units not necessary), then another 2 years from 50 to 25. After 4 years, you’re only at 25% of where you started.
So, we basically *$5^!! ourselves by saying that fat was bad, so “good” fats like butter and tallow and ghee (all from a cow, which has less PUFA no matter what it eats) were verboten, then adding in oils as being “good” instead. And we created an environment where PUFAs can flourish.
Meanwhile, if you read Hyperlipid’s Protons Theory parts, he lays out a very scientific theory that seed oils cause mice (mainly) to overeat and saturated fat to cause lower weight mice. So good, that I was 100% convinced I would eat saturated fat, and I would get full.
But even with my tests, where eating saturated fat did not equate with feeling full, perhaps they were invalid, as I still had fat cells that were filled with PUFAs? Impossible to say, since I can’t get that tested.
For those who argue the amount of PUFAs cause obesity, that seems to be correlation, not causation, though Hyperlipid does provide a possible mechanism for how that could be.
Personally, I can’t rule out that PUFAs do cause obesity, as I can’t test this well enough. We’d need more studies in humans, and there are precious few of those.
It has been awhile since I have done the research but I have urged family members to give up seed oils! First of all, conventional Canola oil is refined with hexane as @PaulL alluded to. That I remember reading.
I never thought of it in terms of one being more satisfying than the other but rather that consuming PUFAs was less healthy than other fats
I just watched a couple of videos by Dr. Anthony Chaffee, a neurosurgeon who has been carnivore for the past three years and off-and-on for the previous twenty. His take, which accords with what I’ve learned, is that PUFA’s in excess, particularly ω-6, and particularly linoleic acid, cause systemic inflammation and insulin resistance. That could eventually lead to obesity, but it doesn’t mean they cause obesity directly.
The other concern with PUFA’s is that the novel types in modern industrial seed oils are replacing some of the fatty acids that are supposed to be in cell walls (all the walls of all cells are made of fatty acids and cholesterol; it’s called a “lipid bilayer”). They render cells more permeable, in ways that are not good for the cell, and they affect how cell linings join together (this can lead, for example, to irritable bowel by making the intestinal lining permeable).
We do need a certain minimal amount of ω-3 and ω-6 fatty acids, but the quantity required is not all that great. Most of us consume far more ω-6 than is good for us. As Stephen Phinney remarks, the challenge, when eating the standard American diet, is not to get enough ω-6, it is to avoid getting too much.
There is apparently some evidence to show that ω-3 fatty acids have beneficial effects, but I don’t know whether that comes from getting them in the correct proportion to ω-6 (they compete for the same receptors, so we should be getting them in pretty much equal amounts), or whether the benefit comes from getting simply enough ω-3 or from getting more than the minimum necessary. I don’t think the researchers have really figured that out yet.
Could not agree more. I would love to eat butter croissants all day long, yum! I would be 50 lbs heavier in 6 months! If I ever decide to cheat again then maybe I will try it, it sounds like a lot of fun but weight loss, that will not happen doing this. Actually now that I think about it, I do not even have to question it. As a teenager who was at her ideal weight I spent a summer with a relative who lived above a bakery in a foreign country, no junk food available. I spent the summer eating freshly baked rye and sourdough and croissants (I would assume although the rye was my favorite), slathered with butter, apricot croissants and all sorts of wonderful treats. By the end of the summer I had a constant stomachache and I had gained 10 lbs! Turns out I found out years later I am somewhat gluten intolerant, I can have some but not at every meal
I am not sure I agree with his premise that PUFAs cause weight gain independent of other consumption. I believe Westman has had some clients lose substantial weight eating almost every meal at McDonalds (consuming only meat and cheese and eggs, no bun) because that was all they could afford and they did not cook. I am sure much of that was fried in canola or other seed oils. Not that anyone recommends doing that, it is not ideal
The combination of reading that the amount of linoleic acid in our diets has increased astronomically (is it a factor of 20,000 or more?), and that we now have a greater concentration of linoleic acid in our cell membranes than previously was enough to have me banish all sources. That’s a seriously scary situation. To think that the structure and function of a membrane is altered … shivers up my spine!
Sorry about being late to this conversation. But the Croissant Diet reminds me of my favorite I like to tell people when they ask. I tell them I am on the B.A.D. Diet. And it is totally Vegan. BAD stands for Beer And Donuts.
Got me through college. How about you?
Actually the advantage of saying this is I don’t have to explain Keto and overcome their preconceived prejudices against Keto. Usually on account of failure at trying it without understanding it, or being scared and misinformed by others.
Mrs Bear and I went fishing. First cast. A fish took the bait. It was silver. They are known as a Skippy. It’s eyes were so big. It looked at us. But didn’t seem to focus. I cut its arteries with a knife. I saw the blood and felt the wild intrinsic. Mrs. Bear cried. We packed up and went home. The beach was beautiful. Fish is a meat that doesn’t change its name in death. Sheep become mutton, cattle become beef. I cleaned, cooked and ate the fish. Mrs Bear demurred. It was delicious. I felt satisfied more so than satiated. We haven’t been fishing since.