What puzzles me as I ruminate on Lean Mass Hyper-Responders (LMHRs):
Given: LDL-P skyrockets shortly after LMHRs go keto.
The science seems clear that dietary cholesterol barely (if at all) affects blood serum cholesterol. (Even Ancel Keys is now said to have agreed with this.)
And yet, the "responder" in “hyper-responder” suggests otherwise.
Put different;y, how could a change in diet from low fat to high fat give rise to both outcomes:
(1) Exert no meaningful effect on LDL, and;
(2) Produce such a remarkable effect on LDL?
There must be a simple way to reconcile these two seemingly contradictory statements, no?