Already showed you that several times and as usual you refuse read so no point bringing that up again?
I know for an absolute unmistakeable FACT that excessive consumption of animal products long-term does cause cancer in human beings as demonstrated and duplicated several times under a microscope.
This is true if you are only consuming meat everyday.
No studies needed Mr. Mark.
Remember reading is fundamental?
If you have ever heard the term blinded by the light of your own ignorance this is a good case of it?
Just because people are not dropping like flies left and right does not mean they don’t already have cancer? It’s a slow process that gradually becomes metastic.
How do you counteract that problem?
The answer might be real folate?
You can’t do that eating only meat (not enough in it when cooked) or man made versions of folic acid?
For example in a Vegan they may not show a Vitamin B-12 deficiency because it is being masked by excessive folic acid or folate but get permanent neurological damage if vitamin B-12 is not replenished fast enough?
Folate deficiencies are not the only reason you get cancer from eating only animal products because eventually you get too many micro-RNA’s floating around in the colon and rectum from amino acid eating bacteria that severely shift the microbiome diversity into a state of putrefaction (putrescine and cadaverine). How is that healthy when you smell like death?
So if I’m purposely eating a certain way that encourages the proliferation of cancer in my gut that is not sane?
Your insulin and glucose levels may look good but there is other damage being created?
References:
[1] “…White rice is fortified with folic acid to prevent folate deficiency. …” …More (i.e. that’s the man made stuff when your constantly and slowly digesting a resistant starch you get a steady supply of non-fortified folate (= longevity/IFG-1 switch turns to off position as well as the cancer proliferation being switched to off position) as well as glucose just like only eating meat?
[2] Folic Acid and Its Metabolites Modulate IGF-I Receptor Gene Expression in Colon Cancer Cells in a p53-dependent Manner