Now, there is no mention of “insulin” in this thread. Let me bring it in 
I’ll have to dig out the talk from low carb down under where this was discussed. As mentioned in another thread:
Protein is insulinogenic for some of us. It seems to be some damage from high glucose or high insulin, because it seems to happen a lot more frequently amoing diabetics. Bottom line, insulin flatlines for most of us if we eat meat on keto, while for a minority a lot of protein is converted to glucose.
So we see a marked insulin response to protein for some people.
Therefore it’s not feasible to compare individual experiences, because the rate of gluconeogenesis may be vastly different between people.
The interesting questions are of course:
- Why do some of us have gluconeogenesis in overdrive? (It does most likely depend on insulin resistance and related damages to the liver, but there may be other factors.)
- Does it matter how you prepare your meat? (Raw or cooked, etc.)
- Does it depend on the type of meat? (Very likely. We know that whey protein is fairly insulinogenic, but these studies were probably done against the background of a western diet so the results may not apply for keto. Even grass fed vs. industrial could be a big difference.)
- Does it matter if we eat 100% carnivore, could 10% non-carnivore food be the difference between active gluconeogenesis or not? (@Fangs looking at you
)
As we have no answers for these questions, all we can do is experiment.
Personally, my 90% carnivore failed. I’m still insulin resistant (last HOMA-IR was 1.7) and I hope my response to protein might change once my insulin response normalizes. I’ll go medium-protein OMAD keto for now, check my HOMA-IR from time to time and I’ll try carnivore again once it’s settled down (below 1 at least). Then I’ll go 100% carnivore (and not 90%) and maybe I can arrange for an insulin measurement after a meal after maybe a month of carnivore. Not sure if it’s a good or bad plan, but it’s my plan and I’ll stick to it 