As as physician for 38 yrs, cardiologist for 30, I have never seen anyone who has not improved their situation by adopting high fat, moderate protein and low carbohydrate.
It is important for people to understand that the medical industry in not about health: http://goo.gl/Blh6rW.
The primary drivers of atherosclerosis, as first published 08/01/1950 http://goo.gl/QMpZiI, have always been the fat carrying proteins, especially the fat delivery LDL-particles: Lower Density (than surrounding water), fat (Lipid in medical jargon) carrying protein particles (which typically transport 3,000-5,000 fat molecules per particle),
Be aware that LDL-C is NOT a measure of LDL particles. its is a number which is not measured from people’s blood sample. It is a guess, Friedewald equation, of how much cholesterol, a fat (made by every cell), is contained within all LDL particles within a 10th of a liter of blood plasma.
This LDL-C value correlates very poorly with actual LDL-particle concentrations, the value which has always most accurately correlated with rates of disease progression and cardiovascular event rates.
The HDL-particles: Higher Density (than surrounding water), fat (Lipid in medical jargon) carrying protein particles (which typically transport no fat, up to a few thousand fat molecules per particle, depending on size) and serve to remove fat molecules (such as in the walls of arteries) from cells overloaded with fats so that they do not die in place (resulting in atherosclerosis).
However, if HDL particles remain small, typical when eating low fat, high carb, HDL particles do not function well. Only the two largest of the 5 groups (by size, density) of HDL particles correlate with lower rates of cardiovascular disease events over time.
Another long promoted lie is that arteries narrow in response to atherosclerosis. Our arteries enlarge: http://goo.gl/xL1roN, http://goo.gl/aRhOU2 http://goo.gl/RgYv8w in compensation which is why, though atherosclerosis, as known from US autopsy studies, from the 1920s on, is typically present in over half the population well before age 10, remains without symptoms for decades until a plaque rupture suddenly shed debris into the blood leading to clots and obstructions downstream (too small to see by any current medical technology, called microvascular disease) and sometime large enough clots to see by angiography.
Because the disease in within the walls of arteries, both angiography &.especially ALL stress tests totally miss the problem until after complications of advanced disease.
Carbs mean sugar and all carbs are only absorbed after digested to no bigger than one or two sugar molecules.
Since, at any given moment, the entire blood stream only contains about a teaspoon of sugar, all excess coming in is rapidly removed by the liver cells, lesser amounts muscle cells, with both have limited capacity for short term storage as glycogen, a carbohydrate.
All excess glucose is converted by the liver into triglyceride fats for storage and shipped out to the body via the fat delivery particles: primarily VLDL & LDL, thus obesity.
After decades of challenges from daily sugar onslaught & rising insulin+amylin outputs to drive sugars into storage, sugar levels rise, eventually enough that the disease industry calls it Diabetes Mellitus, defined as a HbA1c of 6.5% of higher. Yet optimal cardiovascular health corresponds to a HbA1c of 5.0% or lower.
Cholesterol has NEVER been the correct issue, is made by EVERY animal cell https://en.wikipedia.org/wiki/Cholesterol but was promoted by the US government (about 2 decades after politicians sold US citizens on having taxpayers pay for medical services, a power grab for money & power) but then found that the costs (as always) grew far beyond “projections”, typical *.gov politician, bureaucrat & CBO BS.
I hope some of the above details will help some people understand some basics of physiology a bit better.
Milton Alvis
http://www.pureheartsmart.com
http://goo.gl/maps/frsO4