So I was looking up squaline recently because it’s added to the retinol serum (sold topically for the face) that I just bought. Both are lipid soluble.
In the first world, retinol (and it’s metabolites) are derived from vitamin A. When applied topically, it helps your skin cells to proliferate through the mTOR pathway (and reduces acne) while increasing collagen production. If you use too much of it topically, it can make your skin flacky.
Typically seen in third world countries, humans who don’t get enough vitamin A in their diet will go blind.
(Hence why we have retinas, pun not intended).
I found this biochemical pathway while researching what squaline is in my ‘serum’.
I wonder if we should be prescribing statins just because it appears to lower cholesterol or is there something else going on?
I am only doubting it because squaline (a precursor to retinol) is apparently inhibited by statins.
Statins are administered orally to reduce cholesterol production.
Carl Franklin may find this interesting 
(Also, yes this is from Wikipedia, so play nicely)
