While poking around looking for another topic entirely, I happened upon a thread where they were discussing fasting to treat IR. @JorgePasada came up with a great (if geeky!) analogy that really explains it well, and why it might take some time for repairs. I decided it would be a good thing for newbies to read, so here it is:
Tim compares IR to a dam with a leak.
I hope this will help you (well, me, too!) KCKO.
“If you want access to the bottom of your lake. Empty the lake, fix the leak, and then pour all the damn water you have into that thing.”
That statement makes sense to me. Truly fast and then truly feast.
An Explanation of Insulin Resistance by Eugene Paul Ledbetter - Lawrence Livermore National Laboratories ”…According to the biochemical literature, an insulin receptor is a protein molecule located in the wall of a cell. The structure of the molecule is such that a smaller molecule called a ligand may attach to it. There are two kinds of ligands: an agonist, which causes the receptor to function, and an antagonist, which does nothing but prevent the agonist from attaching to the receptor.
Without an attached ligand, an insulin receptor cannot allow glucose molecules to pass from the blood into a cell. An insulin molecule is an agonist, and when one becomes a ligand, an insulin receptor can allow glucose molecules to pass into a cell. But the hormone cortisol is an insulin antagonist, and when a cortisol molecule becomes a ligand, it prevents an insulin molecule from becoming a ligand, which prevents glucose from passing from the blood into the cell involved.
I believe that the action of cortisol as an insulin antagonist is a reasonable explanation of the condition that we call insulin resistance, in which case there is nothing wrong with the insulin receptors or the insulin. Insulin resistance, then, would be part of the natural functioning of an insulin receptor. …” …More
I think what is being described above is because of Chromium and Vanadium deficiency and quality of soil in agriculture. As Dr. Peter Glidden puts it “…”Insulin (can) sticks to the surface of cells and receptors…” (paraphrased). I imagine he is referring to the notion after it is cleared hepatically or muscularly or something to that effect? i.e. residule insulin
I am trying to access the link you posted re: Insulin Resistance but it says I dont have access?
Not sure if you were talking to @atomicspacebunny or me, but if you can’t do something here the first day or two, it’s because the forum watches to see if you are “friend or foe,” so to speak. The more you interact, the more access you’ll have. I should think you could follow links, though. It also helps if you go through the silly bot exercises. 
Just found this forum. Grateful.