He says (about 3 minutes in, it’s only 3:39 long) that if you ever hear someone say there is physiological insulin resistance in the context of a keto diet, you’re supposed to laugh in their face. He thinks this only happens in pregnancy or when you’re a teen.
I guess morning blood sugar rise is “glucose sparing” then?
Personally, I’ve given up using the term “insulin resistant”. I no longer know what it means. I have too many questions.
For instance, I get that there can be a whole-body “insulin resistance” in the sense that your body has to produce more insulin for the same effect as before there was “insulin resistance”, but I’m making an effort to say this is a whole-body insulin resistance. But trying to drill down deeper than this, and the term “insulin resistance” seems to lose meaning. Exactly what is insulin resistant? Fat cells? Muscle cells? Both?
And there could be times when “insulin resistance” is GOOD. For example, the idea behind the theory that saturated fat causes you to not be hungry is because fat cells become – insulin resistant – due to saturated fat intake, thereby throwing up a signal indicating “I’m full.”
And I’m sure muscle cells become more insulin sensitive after working out. See here, for instance:
But this seems to be a transitory effect.
And I wonder why some people can get back to their teenage or near teenage level of fat, whereas some of (like me) can’t. Are our fat cells so insulin resistant, still after 9 years keto? Or is there some other reason?
Anyway, I thought this presentation by Dr. Bikman was interesting. As always, though, I have more questions than answers.