Looking generally good to me. From what I’ve learned in relation to CVD and as a non-doc/sci, most signs seem to point to repeated high-levels of insulin/glucose in the system over time and small dense particle LDL as the best determining factors.
It took my readings about a year to look closer to my preferred mark. The first NMR Lipoprotein test couldn’t even get some readings. On your post, I’m reading HDL went up, good. TG went down, good as far as I can remember. HbA1C down is good. LDL up slightly… question there is how much of that is small very-low-density particle LDL? An NMR Lipoprotein test can get you a pulse on that. It’s about $120 and insurance may cover. Older thinking was that LDL was always bad and the root, period. Evidence has been coming to light over the past few years that implies that was not entirely correct. The jury is still out though. That is just part of the territory right now and a risk that has to be weighed for yourself.
My understanding is that the, smaller, very-low-density pattern B LDL particles get into nooks in the arterial walls more easily. Larger-fluffier pattern A LDL particles don’t tend to get down in those nooks and may be more benign overall. Once the low density LDL gets in the nooks, it’s hard for the body to get rid of them. So the logic goes: if you have more very-low-density particle LDL, there’s more to get in the nooks, therefore less chance the body can get rid of them before more are piled in, when a lot gets in there then you have plaque. If the plaque hits the bloodstream, you get a clot/bad situation.
When assessing my own risk, I look at the HDL vs LDL ratio and then I look at the VLDL particle count from the NMR Lipoprotein results. My aim is always good ratio and then small count of VLDL. I don’t worry so much about the fluffier LDL if the HDL vs LDL ratio looks good. If diabetic, like me, definitely keep an eye on it with an NMR. Diabetes affects how the liver processes the fats which then can affect the LDL towards CVD. Also, 80% of the cholesterol, your liver produces, and it’s not from what you eat. The rub is VLDL is entirely created from the liver (https://www.health.harvard.edu/heart-health/how-its-made-cholesterol-production-in-your-body), so I’m not really sure how much we can control directly from eating more mono/poly vs saturated fat.
As for the fam, who studies the ketosis more, you or them? Follow the data and results yourself. Opines are cheap, healthcare is not.