LMHR data drop

(Michael) #1
(Bob M) #2

The most shocking statement (for most people, particularly doctors) is that LDL level had no relationship to plaque.

(Geoffrey) #3

The doctors I follow have been saying this or similar things for years.
Glad to see more truth is coming out.

Now wait for big pharma to try and shut this down.

(Bob M) #4

For my last lipid test, I got an LDL of 130. My LDL is normally below 100. My cardiologist told me to “watch my diet” because of the high LDL. And I also have a CAC score of zero, so a statin wouldn’t be useful anyway. (And I think the “high” LDL was caused because I was sick. I never changed my diet from the last test. )

I would say 99.999999% of doctors believe whatever the idiots at the American Heart Association tell them.

Agree that the people who sell all of these LDL-lowering drugs are going to be freaking out.

(Bob M) #5

Another thought I had. According to science, if a theory fails once, it fails. They say there’s a person with verified 500+ LDL for 5 years, yet zero plaque. In my mind, that means the idea that LDL causes plaque formation fails. It’s wrong.

But no one seems to think this way.

I was listening to Peter Attia’s interview with Dr. Lipid. Attia said he had a client with extremely high LDL yet with a zero CAC scan score. Meaning, basically no atherosclerosis. A minute or so later, they said “but we know LDL causes atherosclerosis”. Ah, what?

(Geoffrey) #6

I know of several doctors that think this way but you are correct that most mainstream doctors don’t. Couldn’t get their bonuses if they did.

(Michael) #7

Here was Daysprings response on twitter:

“LMHR is a meaningless, made up term by folks with no CV education, that has no scientific definition. In the study abstract there is no mention of any criteria used to define LMHR on those entering the study. Were DEXAs performed to seek lean mass?”

And previously

“Why would anyone care what those with zero lipid/lipoprotein credentials, publications, prestigious teaching or lecture lists or clinical training or licensure spouts. Internet babbling and podcasting are not credentials.”

Tom Dayspring is officially anti-science and is never worth listening too about anything again. I guess spending your whole life working on lipids as causal might seem like a wasted life once they realize it is not in fact causal. Why would anyone respect an expert who instantly uses credential shaming instead of addressing the science. He reminds me of the lead lipidologoist in Canada - Ruth McPhereson. Dogmatic , narcissistic, and anti-science while being (apparently) a scientific leader. Sad state of affairs.

(You've tried everything else; why not try bacon?) #8

Last time my cholesterol was checked, it was 148, and the “healthy” limit was 150. LDL of 130 sounds good to me!

I understand they’ve moved the limit to around 90 and are trying to get it even lower.

(Geoffrey) #9

My LDLc is at 225 and I couldn’t care less.

(You've tried everything else; why not try bacon?) #10

My current doctor won’t test my lipids. I finally figured out why: he knows I’m keto/carnivore, and he doesn’t want to have to go through the statin conversation. My previous doctor was a keto advocate, and they were good friends, so he’s not opposed to my eating this way, but on the other hand, I gather he’s not as enthusiastic about it as his predecessor was.

(Bob M) #11

I met Dave Feldman at the last Keto Fest. He said that his study might bring Peter Attia around, but he didn’t think Dayspring would ever come around.

I mean, if you go by the moniker “Dr. Lipid”, why would you? :grinning:

I am concerned that the statin/PCSK9 manufacturers might freak out about this stuff.

(You've tried everything else; why not try bacon?) #12

“It is difficult to get a man to understand something, when his salary depends on his not understanding it.”—Upton Sinclair

(Michael) #13

An excellent job by Nick, but also Dave at trying to rein Ken in. https://m.youtube.com/watch?v=rL9uabr-TeA

(Bob M) #14

That’s definitely true.

He (Dayspring) actually personal messaged me when I was on Twitter (a while ago, quit after Musk bought it), telling me how bad Lp(a) was for me.

The problem is that my Lp(a) is so high that I’d have to have a drop of 70% or more to get to “normal” range. There is nothing that will drop my Lp(a) that much.

The other problem is that I have none of the effects for atherosclerosis that high Lp(a) supposedly causes. So, why would I be concerned about something that causes an issue that isn’t happening?

Lp(a) could cause coagulation issues, but no one seems concerned about those. Not sure why. And I don’t know how to address these other than to have a low “inflammation” diet, which to me is keto/low carb with some fasting.