What we are trying to promote is a more nuanced understanding of how the types of food eaten determine whether food will be stored or metabolized, depending on insulin level. (Insulin is the body’s fat-storage hormone, plus it has other deleterious effects on the body, relating to inflammation and the suppression of anti-oxidants.)
It has also been shown that the body matches its energy expenditure to its intake, so eating at a caloric deficit causes the basal metabolic rate to go down, and prevents excess stored fat from being metabolized, even if carbohydrate intake is low enough not to increase insulin secretion. By contrast, eating a sufficient amount of food (as, for example, by eating to satiety) encourages the body to raise its metabolic rate and to mobilize excess fat for metabolism (in the context of low carbohydrate, of course).
Eating to satiety rather than to a pre-determined caloric level encourages the bodily mechanisms to adjust appetite to a level that permits the metabolism of both food intake and also excess stored fat (again, assuming carbohydrate is kept low).
This is the rationale for a well-formulated ketogenic diet. Does it make sense to you?